京都大学 · 生命科学研究科

研究キーワードaldehyde degradation deficiency syndrome・Fanconi anemia-like syndrome・inherited bone marrow failure・aldehyde metabolism・patient-derived iPSCs・aldehyde detoxification

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• p53 directly regulates the transcription of the human frataxin gene and its lack of regulation in tumor cells decreases the utilization of mitochondrial ironGene,
• The role of SLFN11 in DNA replication stress response and its implications for the Fanconi anemia pathwayDNA Repair,
• Neurotransmitter transporter family including SLC6A6 and SLC6A13 contributes to the 5-aminolevulinic acid (ALA)-induced accumulation of protoporphyrin IX and photodamage, through uptake of ALA by cancerous cellsPhotochemistry and Photobiology,
• The ribonuclease domain function is dispensable for SLFN11 to mediate cell fate decision during replication stress responseGenes to Cells,

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